The objective of the present study was to determine median age at menarche and the influence of familial instability on maturation. The sample included 7047 girls between the ages of 9 and 17 years from Tuzla Canton. The girls were divided into two groups. Group A (N=5230) comprised girls who lived in families free of strong traumatic events. Group B (N=1817) included girls whose family dysfunction exposed them to prolonged distress. Probit analysis was performed to estimate mean menarcheal age using the Probit procedure of SAS package. The mean menarcheal age calculated by probit analysis for all the girls studied was 13.07 years. In girls from dysfunctional families a very clear shift toward earlier maturation was observed. The mean age at menarche for group B was 13.0 years, which was significantly lower that that for group A, 13.11 years (t=2.92, P<0.01). The results surveyed here lead to the conclusion that girls from dysfunctional families mature not later but even earlier than girls from normal families. This supports the hypothesis that stressful childhood life events accelerate maturation of girls.

A study of the age at menarche was conducted in Tuzla Canton on a sample which included 7047 girls between the ages of 9 and 17 years. Data were collected using thc status quo method. Median age at menarche estimated by Probit analysis was 13.07±0,05 years with a standard deviation of 1,05 years. Girls in rural places had a delay in their menstruation, with a mean age at menarche of 13,19 years, compared to those who lived in urban places, with a mean age at menarche of 12,84 years (P < 0.0001). As no previous Information is available about the age at menarche in Tuzla Canton, the present results will afford a basis for future studies which should aim at analyzing the secular trend in menarche while attempting to define the differences between the various socio-economic levels.

In the paper it was identified firstly the short stature, then importance of anamnesis, physical examination, anthropometric measurements and calculated parameters of growth. Then followed the classification of causes for short stature with a specific review on differentiation of normal from pathologic variants. Further in the text it was pointed out the importance of observing normal variants of short stature since between 84% and 87% children are with height under the 3rd percentile. Then followed tables showing pathologic causes of the short stature, and then diagnostic and therapeutic approach to the child with proportionately short stature and short stature associated with dysmorphic features.